Sinoe Medical Association USMLE Tutoring and reviews

09/04/2026

https://youtu.be/ghXCPLOKnD4

Polycythemia Vera for USMLE Step 1 & Step 2 CK. In this high-yield ...

09/04/2026

https://youtube.com/shorts/7Aun7_bS-uE?feature=share

Hairy cell leukemia is a rare, indolent B-cell malignancy with seve...

09/04/2026

https://youtu.be/zL0MmvjXLNc

Leukenias case series

like, comment subscribe for more Hairy Cell Leukemia: The "Dry Ta...

09/04/2026

Adrenal gland layers and hormones
Elevate Your Score with Sinoe Medical Association:

FREE "Top 50 USMLE Buzzwords" Guide: https://sinoemedicalassociation.org/usmle
Join the Sinoe Insider Membership ($15/mo): Access our full lecture vault.
Book 1-on-1 MD-Led Tutoring ($60/hr)

09/04/2026

hemostasis in few second explanation
Master the Coagulation Cascade for USMLE Step 1, Step 2 CK, and NCLEX. We move beyond the "factor soup" to teach you the MD-led logic of the Intrinsic, Extrinsic, and Common pathways. Led by Dr. Danil Hammoudi, this review focus on the diagnostic signals you need to decode bleeding disorders in seconds.

What we master in this High-Yield Review:

The "3-7" Rule: Why the Extrinsic pathway is the fastest way to the answer.
PT vs. PTT Secrets: Connecting the pathways to Warfarin, Heparin, and the factors they monitor.
The Common Pathway: Mastering the 10, 5, 2, 1 "Countdown."
Clinical Traps: Identifying Hemophilia, von Willebrand Disease, and Vitamin K deficiency vignettes.
Pharmacology Links: Where DOACs, LMWH, and Vitamin K antagonists actually hit the cascade.

09/03/2026

Coagulation Cascade

09/03/2026

https://youtube.com/shorts/C1i-8_43orY?feature=share
coagulation cascade
Master the Coagulation Cascade for USMLE Step 1, Step 2 CK, and NCLEX. We move beyond the "factor soup" to teach you the MD-led logic of the Intrinsic, Extrinsic, and Common pathways. Led by Dr. Danil Hammoudi, this review focus on the diagnostic signals you need to decode bleeding disorders in seconds.

What we master in this High-Yield Review:

The "3-7" Rule: Why the Extrinsic pathway is the fastest way to the answer.
PT vs. PTT Secrets: Connecting the pathways to Warfarin, Heparin, and the factors they monitor.
The Common Pathway: Mastering the 10, 5, 2, 1 "Countdown."
Clinical Traps: Identifying Hemophilia, von Willebrand Disease, and Vitamin K deficiency vignettes.
Pharmacology Links: Where DOACs, LMWH, and Vitamin K antagonists actually hit the cascade.
🚀 Elevate Your Score with Sinoe Medical Association:

FREE "Top 50 USMLE Buzzwords" Guide: https://sinoemedicalassociation.org/usmle
Join the Sinoe Insider Membership ($15/mo): Access our full lecture vault.

Success on the USMLE Step 1 and Step 2 requires more than just rote memorization; it requires a deep understanding of how to think like a physician. Led by Dr. Danil Hammoudi, our association offers a unique, integrated approach to medical education. 30 Years of Excellence: Decades of dedication to....

08/28/2026

https://youtube.com/shorts/MTtkYSFl2EQ?feature=share
Feeling exhausted, weak, dizzy, or short of breath? Anemia occurs when the blood has too few functional red blood cells or insufficient hemoglobin to deliver oxygen effectively.

In this 60-second health fact, discover:

Common symptoms of anemia
The importance of hemoglobin and oxygen delivery
Microcytic, normocytic, and macrocytic anemia
Frequent causes, including iron deficiency, blood loss, chronic disease, hemolysis, vitamin B12 deficiency, and folate deficiency
Why anemia is a diagnosis that requires identifying the underlying cause

A complete blood count, MCV, reticulocyte count, ferritin, iron studies, vitamin B12, folate, and additional targeted tests can help establish the diagnosis.

Important: Do not assume every anemia is caused by iron deficiency. Taking iron without confirming the cause may delay the correct diagnosis.

Presented by Sinoe Medical Association
Danil Hammoudi, MD & Imene Benayache, MD

This video is for education only and does not replace professional medical evaluation.

08/28/2026

https://youtube.com/shorts/sljiMwec7rw?feature=share

What happens when blood calcium begins to rise—and how does calcitonin help bring it down?

Produced by thyroid parafollicular C cells, calcitonin acts as a physiologic brake on elevated serum calcium. It inhibits osteoclast-mediated bone resorption, reduces calcium release from bone, and promotes renal calcium excretion. However, its calcium-regulating role in adults is much weaker than that of parathyroid hormone and vitamin D.

This cinematic medical review explains:

Calcium distribution and physiologic functions
Ionized versus total serum calcium
Albumin’s effect on measured calcium
Corrected calcium and when to measure ionized calcium
Calcitonin synthesis by thyroid C cells
Calcitonin’s effects on osteoclasts, bone, and kidneys
PTH, calcitriol, and calcitonin interactions
The difference between calcitonin and calcitriol
Clinical use of calcitonin in hypercalcemia and Paget disease
Tachyphylaxis with repeated calcitonin administration
Calcitonin as a tumor marker for medullary thyroid carcinoma
MEN2, RET mutations, and associated endocrine tumors
High-yield USMLE and board-exam traps
High-yield distinction
Calcitonin: lowers serum calcium, primarily by inhibiting osteoclasts
Calcitriol: increases intestinal absorption of calcium and phosphate
PTH: increases serum calcium while generally lowering serum phosphate

Calcitonin is pharmacologically useful, but it is not the principal regulator of calcium homeostasis in adults.

Presented by Sinoe Medical Association
Danil Hammoudi, MD & Imene Benayache, MD

This video is intended for medical education and does not replace professional medical advice.

08/28/2026

https://open.spotify.com/episode/5EL4c7iZbsMXrPs1YYQvBo?si=J45jq0UJTSCLLOij6ZAgFg

Iodine induced thyrotoxicosis and_Wolff-Chaikoff effects

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