Elle Lipedema Fit

Elle Lipedema Fit Lipedema Coach BHSc | Complex Bodies
Helping you train without flares
FREE Legs Framework 👇

07/09/2026

Theres something going on in the Lipedema Community online right now that needs to be addressed.

An influx of fake AI content creators stealing bodies and likeness from then using this to sell guides to make money. Add insult to injury these people buy original guides from these women then go and illegally on sell them - the lion the witch and the audacity.

The craziest thing too is people are falling for and actually believe these people are real. Do me a favour and report them - lets keep our community safe.

Elle James

07/09/2026

I coach women with all stages of lipedema and hypermobility with intelligent, capacity-based training.

We build strength within what your joints, tissue, and recovery can handle today - so your capacity grows without constant flares, pain, or burnout.

This is 1:1, not a template. I program around your stage, your symptoms, your history.

DM me CAPACITY and I’ll send you how my 1:1 online coaching works.

06/09/2026

Happy Fathers Day to my biggest supporter my dad Nick Hemming 💜💕

05/09/2026

If you have Lipedema, you’ve probably tried to whole eat less and move more thing too many times to count.
But no one told you the real key to success is lowering insulin.
When insulin is high all day, your body gets the signal to STORE. To hold onto fluid, to hold onto fat, especially in your lower body.
When insulin comes down, your body finally feels safe enough to let go.

Here’s where to start:
1. Carbs LAST
2. 30-40g real protein per meal
3. Build your glucose sink (muscle)
4. Walk straight after meals
5. 8+ hours sleep
6. Manage stress / stop grazing

Save this and pick ONE to start this week. Which one will it be?

Lipedema

03/09/2026

The difference between a normal coach and a lipedema coach?
A normal coach sees missed workouts or low motivation.

A lipedema coach sees:
→ A body that needs a deep understanding of lipedema to train effectively
→ A client whose capacity changes daily, and that’s normal
→ Flares that need a management plan, not punishment
→ A woman who needs to feel seen, not judged

I coach this way because I’ve lived it and I know what it’s like to do everything “right” and still have your body feel wrong.

That’s why my clients stay and thats why they all get results. My clients learn how to improve their wellbeing, grow muscle and reframe how they relate to Lipedema longterm.

The lipedema genetics conversation is changing 👀🧬Not just “what genes did you inherit?” but “what’s happening inside the...
31/08/2026

The lipedema genetics conversation is changing 👀🧬

Not just “what genes did you inherit?” but “what’s happening inside the affected tissue?”
That’s the idea behind somatic mosaicism - when a genetic change happens after fertilization, so only SOME cells carry it. Blood can look normal while affected tissue tells a different story.
This isn’t proven in lipedema - it’s an emerging hypothesis researchers are starting to explore. Lipedema is currently understood as polygenic/complex, not caused by one single gene.
I used the case of Mandy Sellars (PIK3CA-related overgrowth spectrum / PROS is not lipedema) in the slides to show how mosaicism can work in overgrowth conditions. Same lesson: sometimes you have to look in the tissue, not just the blood.
Fascinating shift, right?

References:
Michelini S, et al. Unraveling lipedema: comprehensive insights and the path to future discoveries. npj Metabolic Health and Disease. 2024.
2. Child AH, et al. Lipedema: An Inherited Condition. Familial clustering suggests genetic etiology but mode is challenging; supports polygenic model with multiple susceptibility genes. ďżź
3. Keppler-Noreuil KM, et al. PIK3CA-related overgrowth spectrum (PROS): Diagnostic and testing criteria. PROS disorders can be caused by mosaic PIK3CA mutations. Example of somatic mosaicism in overgrowth. ďżź
4. Luks VL, et al. Case reports: Whole-exome sequencing on resected tissue revealed PIK3CA c.3140A>G with 11% mosaicism leading to PROS diagnosis (CLOVES). Illustrates tissue-specific mosaicism detection. ďżź

Yes your estrogen can be LOW on a blood test… and that still doesn’t tell you everything about what’s happening in your ...
24/08/2026

Yes your estrogen can be LOW on a blood test… and that still doesn’t tell you everything about what’s happening in your tissues.

This is where the conversation around lipedema and hormones gets much more interesting as gynoid adipose tissue isn’t simply passive storage. It is metabolically active tissue that can participate in steroid hormone production, conversion and metabolism.

And your estrogen status is influenced by more than the number printed on one serum blood test, we also have to consider:

→ local aromatase activity
→ steroid-converting enzymes
→ genetic variation
→ liver conjugation
→ gut microbiome activity
→ enterohepatic recycling
→ bowel regularity
→ HRT, if applicable
→ and the individual hormonal environment of the tissue

This does NOT mean that low serum estrogen = “estrogen dominance.” And it certainly doesn’t mean every woman with lipedema needs to “detox estrogen.”

It means we need to stop treating one blood marker as the complete picture because it isn’t. Especially when we’re talking about a condition that has such a strong sex-hormone-associated, gynoid distribution.

So the real goal isn’t to chase estrogen lower, it is to understand the system because lipedema is complex and your biology deserves more than a single number on a lab report.

Educational content only. Not medical advice.

DM CLEARANCE if you are interested in my coaching waitlist or see Lindsay Huelse | Weight Loss for Moms for in-depth lab analysis like DUTCH

22/08/2026

Why are scientists suddenly so interested in one gene - AKR1C1 - for lipedema? 🧬

Well, for decades lipedema was dismissed as “just fat” or a lifestyle issue.
Then in 2020 they found the first mutated gene linked to non-syndromic lipedema in one family - 3 women with autosomal dominant lipedema all shared an AKR1C1 variant. AKR1C1 is the off-switch that turns active progesterone into its inactive form inside the fat tissue.
Their variant only worked at ∟50%. So active progesterone hung around longer in the subcutaneous fat and kept the fat-storage machinery switched ON.
This is why researchers are excited and why that line in thos reel states “pointing to new therapeutic targets BEYOND diet and exercise”:
1. Because it explains the pattern - 11% of women, female predominance, onset at puberty / pregnancy / menopause - it was always hormonal. This shows it’s about how fat tissue metabolizes hormones locally, not just blood levels.
2. It’s not just one family anymore either, in late 2023 they found AKR1C2 (the second gene) overexpressed in 24% of patients.

The BIG 2026 reviews now call the whole AKR1C pathway a central biological pathway for lipedema. Plus 3 common polymorphisms linked to higher risk.

3. It basically explains why diet and exercise don’t fix it, because it’s a pre-receptor metabolism + genetics + environment.
4. For us, this opens more doors.. if it’s hormone metabolism, then we can finally look at biomarkers, targeted therapies, personalized approaches, and why mental health is now considered a CORE part of lipedema, not just a consequence of living with it - because these same enzymes make neurosteroids 👀

That’s why 19 countries just agreed in the first international consensus: it’s never just an abnormal fat condition.
This doesn’t mean low progesterone or that HRT will fix or worsen lipedema - that wasn’t tested. It means hormone metabolism matters, and that’s why research beyond compression and liposuction is finally happening.

Educational only. Save this for your clinician appointment.

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